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Are BTK and PLCG2 mutations essential and sufficient for ibrutinib resistance in Persistent lymphocytic leukemia?
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44 Moreover, anergic cells Commonly keep the next susceptibility to apoptosis Except if anti-apoptotic proteins such as BCL2 are overexpressed, as is the case for CLL cells.forty five In truth, most key therapeutic innovations developing in the final 10 years are connected to the inhibition of BCR and BCL2-mediated signaling.
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